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Post Info TOPIC: Everything you need to know about Anavar


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Everything you need to know about Anavar
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Everything you need to know about Anavar...

MYTHS Myth #1 - Anavar will not suppress the HPTA. False. Anavar, used in adequate dosages, will shut you down. To what degree you experience side effects of suppression (loss of libido, lethargy) is entirely dependent upon the individual and the dosages used.

Myth #2 - Var is a weak anabolic, and is not effective unless stacked with a more androgenic compound. This could not be further from the truth. At dosages of 40mg a day and higher, anavar is incredibly effective at adding water free LBM. At around day 6-7, increased vascularity should become apparent (assuming your oxandrolone is legitimate in its dosing), and strength gains should start appearing around day 14. If used during a clean bulk, gains of 10-20 pounds are possible. If cutting, you will maintain weight, or even put on 5-10 pounds (depending on the rate of fat loss/severity of diet). You will keep all of your gains with proper PCT.

Myth #3 - Anavar will not require any type of PCT. This is one ive never understood. It's a pretty commonly known fact now that var is a suppressive compound. So why is it that some individuals still refuse to make a small investment in some clomid/nolva....this is your testicular function we're talking about. That said, PCT required for var is not as "heavy" as PCT for, say, a test/eq cycle. 15-20 days @ 50mg clomid should be sufficient. LIBIDO The only real issue of concern that i have found when running anavar alone is slight libido suppression. Anavar is suppressive enough to where you WILL feel a difference in your sex drive (and not for the better ) when using 40+mg a day.

There are three options to counteract this. #1 - Tribulus + Avena Sativa - Trib at 4-7g a day and Avena Sativa at 3-4g a day tend to help prevent any loss in performance or ability to get it up. However, using effective dosages is going to end up being as or more expensive than options 2 or 3...but its your call.

#2 - Proviron - If hairloss is an issue in your choice to use anavar, then you may want to avoid this one. But 25mg ED proviron, starting after week 2, will keep you rock hard. And it will help to harden up your muscles too .

#3 - Maintenance Test Dosage - Finally, you could choose to use testosterone to keep your willy in shape. At a dosage of around 200mg, split bi weekly, everything should keep running smoothly. Also, this will contribute to your gains much moreso than than options 1 or 2. I would keep nolva onhand on the off chance that you are severely gyno prone. Bloating should not be an issue at this dosage.

BENEFITS Anavar is a badass drug. This is why.

#1 - Vascularity Oxandrolone will make you veiny as all hell. And quickly. Look out for brand new bulging forearms veins by around day 6. If you are following a cutting regimen, expect new spider webs in your chest, shoulders and quads by around day 21.

#2 - Pumps When on var, the pumps are constant. Bored sitting in class/at work? Do some unweighted calf raises. After about three minutes, your calves will be ready to pop. Youll be doing something like drinking a cup of water, and after a minute of holding it, your bi will be completely full and pumped. You may have to cut some sets short in the gym due to the painful pumpage.

#3 - Strength Even when cutting, you can expect new strength gains every workout after about day 14-21.

#4 - Fat Loss Anavar has been shown to contribute to accelerated fat loss in both subcutaneous and visceral fat, concentrated effects in the abdomen and thigh area. And if youve used the drug, you can attest to this...if you cant sport the 6-8 pack look on var, its not gonna happen . CYCLE Anavar should be run @ at least 40mg a day to see all of the benefits it offers. Dosages upwards of 80mg have been shown to exhibit diminishing returns. Also, i cant imagine the intensity of the pumps at that kind of dosage. LIVER PROTECTION Anavar is a 17 Alpha Alkylated steroid, and is toxic. It has been shown to be less toxic than other orals, and is even used as liver treatment for recovering alcoholics. Still, i would limit my time using it to 8 weeks, 10 at the most. It would be beneficial to you liver to use several different OTC supplements during, and perhaps after your cycle. A few preventive measures never hurt anyone . 1 - Milk ThistleThe classic liver protectant herb.supposedly works by blocking the entrance of harmful substances to liver cells, and hastening their expulsion. Make sure there is a high standardization of Silymarin 2 - R ALA A powerful antioxidant 3 - NAC Supports liver function and production of l-glutathione 4 - Vitamin C and E Antioxidants 5 - LOADS of water Helps to flush out your entire system

LIPID PROTECTION Anavar isnt going to kill your cholesterol levels like some drugs (winny being one of the worst), but it may put your LDL/HDL profiles outside of the normal range. There are a few things that help, but as long as your not using 60+mg daily or running it for more than 10 weeks, i would just use flax... 1 - Flax Oil Consuming lots of essential fatty acids promotes overall health, as well as helping to keep your lipid profile from becoming too bad. 2 - Policosanol Used at 20mg daily to keep your HDL (good cholesterol) levels from crashing, and your LDL from becoming too high. 3 - Niacin Preferably the flush free variety. If you wish, niacin can be used at 1-2g ED for a short period post-cycle to normalize HDL levels. Do not use for more than 7-14 days, as liver toxicity can be an issue when using those dosages of niacin for long periods of time Oxandrolone and fat burning Oral anabolic steroid treatment, but not parenteral androgen treatment, decreases abdominal fat in obese, older men. Lovejoy JC, Bray GA, Greeson CS, Klemperer M, Morris J, Partington C, Tulley R. Pennington Biomedical Research Center, Baton Rouge, Louisiana 70808-4124, USA. OBJECTIVE: To compare the effects of testosterone enanthate (TE), anabolic steroid (AS) or placebo (PL) on regional fat distribution and health risk factors in obese middle-aged men undergoing weight loss by dietary means. DESIGN: Randomized, double-blind, placebo-controlled clinical trial, carried out for 9 months with primary assessments at 3 month intervals. Due to adverse blood lipid changes, the AS group was switched from oral oxandrolone (ASOX) to parenteral nandrolone decaoate (ASND) after the 3 month assessment point. SUBJECTS: Thirty healthy, obese men, aged 40-60 years, with serum testosterone (T) levels in the low-normal range (2-5 ng/mL).

MAIN OUTCOME MEASURES: Abdominal fat distribution and thigh muscle volume by CT scan, body composition by dual energy X-ray absorptiometry (DEXA), insulin sensitivity by the Minimal Model method, blood lipids, blood chemistry, blood pressure, thyroid hormones and urological parameters. RESULTS: After 3 months, there was a significantly greater decrease in subcutaneous (SQ) abdominal fat in the ASOX group compared to the TE and PL groups although body weight changes did not differ by treatment group. There was also a tendency for the ASOX group to exhibit greater losses in visceral fat, and the absolute level of visceral fat in this group was significantly lower at 3 months than in the TE and PL groups. There were significant main effects of treatment at 3 months on serum T and free T (increased in the TE group and decreased in the ASOX group) and on thyroid hormone parameters (T4 and T3 resin uptake significantly decreased in the ASOX group compared with the other two groups). There was a significant decrease in HDL-C, and increase in LDL-C in the ASOX group, which led to their being switched to the parenteral nandrolone decanoate (ASND) after 3 months. ASND had opposite effects on visceral fat from ASOX, producing a significant increase from 3 to 9 months while continuing to decrease SQ abdominal fat. ASND treatment also decreased thigh muscle area, while ASOX treatment increased high muscle. ASND reversed the effects of ASOX on lipoproteins and thyroid hormones. The previously reported effect of T to decrease visceral fat was not observed, in fact, visceral fat in the TE group increased slightly from 3 to 9 months, although SQ fat continued to decrease. Neither TE nor AS treatment resulted in any change in urologic parameters.

CONCLUSIONS: Oral oxandrolone decreased SQ abdominal fat more than TE or weight loss alone and also tended to produce favorable changes in visceral fat. TE and ASND injections given every 2 weeks had similar effects to weight loss alone on regional body fat. Most of the beneficial effects observed on metabolic and cardiovascular risk factors were due to weight loss per se. These results suggest that SQ and visceral abdominal fat can be independently modulated by androgens and that at least some anabolic steroids are capable of influencing abdominal fat. It has been postulated that Oxandrolone is especially good at reducing visceral fat due to its high AR binding affinity. It appears to be better at binding to the AR (at even amounts) then Test or deca . It would make sense if it is working through the AR if it also increases AR expression in adipose tissue as well (upregulating the AR in adipose tissue adding to lipolytic effects), which the following study seems to show: Short-term oxandrolone administration stimulates net muscle protein synthesis in young men. Sheffield-Moore M, Urban RJ, Wolf SE, Jiang J, Catlin DH, Herndon DN, Wolfe RR, Ferrando AA. Department of Surgery, University of Texas Medical Branch, and Shriners Burn Hospital for Children, Galveston 77550, USA. melmoore@utmb.edu Short term administration of testosterone stimulates net protein synthesis in healthy men. We investigated whether oxandrolone [Oxandrin (OX)], a synthetic analog of testosterone, would improve net muscle protein synthesis and transport of amino acids across the leg. Six healthy men [22+/-1 (+/-SE) yr] were studied in the postabsorptive state before and after 5 days of oral OX (15 mg/day). Muscle protein synthesis and breakdown were determined by a three-compartment model using stable isotopic data obtained from femoral arterio-venous sampling and muscle biopsy. The precursor-product method was used to determine muscle protein fractional synthetic rates. Fractional breakdown rates were also directly calculated. Total messenger ribonucleic acid (mRNA) concentrations of skeletal muscle insulin-like growth factor I and androgen receptor (AR) were determined using RT-PCR. Model-derived muscle protein synthesis increased from 53.5+/-3 to 68.3+/-5 (mean+/-SE) nmol/min.100 mL/leg



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